Mitophagy Mechanism and Its Relationship With Diseases
| dc.contributor.author | Ceylan, Canan | |
| dc.contributor.author | Dönmez, Nurcan | |
| dc.contributor.author | Ceylan, Canan | |
| dc.date.accessioned | 2025-07-07T11:40:07Z | |
| dc.date.available | 2025-07-07T11:40:07Z | |
| dc.date.issued | 2025 | en_US |
| dc.department | Spor Bilimleri Fakültesi | en_US |
| dc.description | Akademik Perspektiften Sağlık Bilimleri / Editör: Dr.Öğr.Üyesi Necati BÜKECİK -- Yaz Yayınları -- E_ISBN 978-625-5838-37-7 --Afyonkarahisar, 2025. | en_US |
| dc.description.abstract | Mitochondria, essential organelles that serve as the cellular energy centers in all eukaryotic cells, are responsible for aerobic respiration and the production of adenosine triphosphate (ATP) through oxidative phosphorylation (Nunnari & Suomalainen, 2012; Özsan, 2023). In addition to being the primary organelles for reactive oxygen species (ROS) production during the electron transport chain, mitochondria are also exposed to oxidative stress under conditions of structural and functional impairment (Özsan, 2023; Wong & Holzbaur, 2014). | en_US |
| dc.identifier.endpage | 52 | en_US |
| dc.identifier.orcid | 0000 0002-0424-4403 | en_US |
| dc.identifier.orcid | 0000-0003-4271-598X | en_US |
| dc.identifier.startpage | 29 | en_US |
| dc.identifier.uri | https://hdl.handle.net/20.500.12436/7803 | |
| dc.institutionauthor | Ceylan, Canan | |
| dc.language.iso | en | |
| dc.publisher | Yaz Yayınları | en_US |
| dc.relation.ispartof | Akademik Perspektiften Sağlık Bilimleri | en_US |
| dc.relation.publicationcategory | Kitap Bölümü - Ulusal | en_US |
| dc.rights | info:eu-repo/semantics/openAccess | en_US |
| dc.title | Mitophagy Mechanism and Its Relationship With Diseases | en_US |
| dc.type | Book Part | |
| dspace.entity.type | Publication | |
| relation.isAuthorOfPublication | 1934d30d-5e97-4c81-abef-fa3c017e570c | |
| relation.isAuthorOfPublication.latestForDiscovery | 1934d30d-5e97-4c81-abef-fa3c017e570c |
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